As we all know, premature ejaculation can be classified in various ways. One classification method divides it into primary premature ejaculation and secondary premature ejaculation. The difficulty of treating primary and secondary premature ejaculation cannot be generalized simply; rather, it depends on multiple factors.

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Primary premature ejaculation typically refers to a condition that has been present since the first sexual experience, often characterized by short sexual duration. Its etiology is relatively complex, including genetic factors, neurobiological factors, psychological factors, and others. Since the cause is often unclear, treatment may require a combination of various methods and may involve prolonged adjustment and experimentation, making it relatively more challenging to treat.

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Secondary premature ejaculation, on the other hand, refers to cases where sexual function was initially normal but later developed due to specific causes, such as urological diseases (most commonly chronic prostatitis), psychological stress, medication side effects, and others. If the underlying cause of secondary premature ejaculation can be clearly identified and effectively addressed, treatment outcomes are often more significant, and it may be relatively easier to treat.

However, whether it is primary or secondary premature ejaculation, treatment effectiveness varies from person to person. Individual differences among patients, such as treatment compliance, psychological state, lifestyle habits, and the level of cooperation from their sexual partners, can all influence treatment outcomes.

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In summary, for the treatment of premature ejaculation, the key is to seek medical attention promptly, undergo accurate diagnosis and evaluation, and then develop a personalized treatment plan.